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G can also modulate various signaling pathways and interact with target proteins

Kyoto Encyclopedia of Genes and Genomes (KEGG)-based analysis showed that the differentially expressed metabolites were involved in arachidonic acid metabolism, the glycerophospholipid pathway, and inflammatory mediator regulation of Transient receptor potential channel (TRP) channels, suggesting that allicin may change the metabolism in the peri-infarct cortex, especially arachidonic acid metabolism (Fig

Journal of Clinical Investigation, 97(8), 1941-1946

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What drives these phases is oxidative stress and inflammation, which, in turn, cause a myriad of aberrant gene expressions in the transformed cell population and surrounding cells of the lesion [60]