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Stressors such as physical exertion may lead to mitochondrial structural damage and functional impairment, resulting in reduced ATP synthesis, an increase in ROS production, and the accumulation of metabolic byproducts, such as lactate

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Despite 80% power to detect a twofold reduction in 8-OHdG status compared to placebo, no significant differences were detected between groups or within group between baseline and postintervention