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Vitamin D prevents hypoxia/reoxygenation-induced blood-brain barrier disruption via vitamin D receptor-mediated NF-kB signaling pathways

While translational challenges in device durability and biocompatibility remain, interdisciplinary advances are paving the way for its clinical implementation, offering a promising pathway toward achieving long-term disease modification in KOA

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They have sufficient supporting information for the FDA to evaluate and do not appear to pose significant safety risks

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