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A Novel Antagonist of the Prostaglandin E(2) EP(4) Receptor Inhibits Th1 Differentiation and Th17 Expansion and Is Orally Active in Arthritis Models

On the other hand, a study led by Towfighi et al

To assess the functional loss of FBXL5, we examined the response of the FBXL5IRP2 axis to changes in cellular iron levels induced by ferric ammonium citrate (FAC) or the iron chelator, desferrioxamine (DFO), in wild-type (WT) and pooled FBXL5 KO cells