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Experimental modulation of endothelial survival, angiogenic signaling and metabolic state reveals that intact tyrosine kinase receptor (Tie2) signaling, glycolytic competence of peritubular capillary endothelial cells, and sustained VEGF-A-vascular endothelial growth factor receptor 2 (VEGFR2) communication from injured TECs are all required to maintain endothelial identity, prevent EndMT and limit microvascular rarefaction [121]

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Ewels, P., Magnusson, M., Lundin, S

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