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Results Serine/glycine pathway metabolites were significantly consumed in response to radiotherapy in NSCLC patients and cell models

This upregulation occurs through the activation of PKC and MAPK signaling pathways, which also suppress eNOS activity, reducing NO bioavailability and exacerbating endothelial dysfunction (103, 104)

The authors detected modulations of the redox status of the cells, without any oxidative damage, by a positive modulation of antioxidant enzyme expression and a significant increase in GSH (glutathione) levels

Activating Intrinsic Neuronal Defense (bottom left, blue): Engaging cellular pathways such as Nrf2 and SIRT1 enhances antioxidant capacity, mitochondrial function, and autophagic clearance

European journal of anaesthesiology , 18 7, 419-22