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However, this is currently determined on a case-by-case basis

This paper describes how mitochondria produce ROS

Background: THAP1 variants are among the earliest identified genetic variations associated with isolated generalised dystonia

N-acetylcysteine attenuates myocardial dysfunction and postischemic injury by restoring caveolin-3/eNOS signaling in diabetic rats

Lifelong voluntary aerobic exercise protocols have been shown to largely prevent age- and western diet-induced vascular endothelial dysfunction and arterial stiffening throughout the lifespan in mice, and this protective effect appears to be mediated by the mitigation of mitochondrial-derived oxidative stress and inflammation [116]